Síguenos en Twitter       Síguenos en Facebook       Síguenos en YouTube       Siguenos en Linkedin       Correo Grupsagessa       Gmail       Yahoo Mail       Dropbox       Instagram       Pinterest       Slack       Google Drive       Print       StumbleUpon NEW

SOBRE EL AUTOR **

Mi foto
FACP. Colegio de médicos de Tarragona Nº 4305520 / fgcapriles@gmail.com

WORLD EMERGENCY MEDICINE SOCIETIES & RELATED

EMERGENCY MEDICINE DAY: MAY 27

Buscar en contenido

Contenido:

jueves, 22 de octubre de 2015

Cirrosis hepática: complicaciones

emDocs - October 21, 2015 - By Long B - Edited by: Robertson J and Koyfman A
"Cirrhosis is the late stage of progressive hepatic fibrosis and is considered irreversible. In 2010, cirrhosis was the 8th leading cause of death in the U.S., and it most commonly due to Hepatitis C, followed by alcohol.1,2 Acute liver failure can also be due to drugs (most commonly acetaminophen), viral hepatitis, autoimmune, ischemia (shock liver), Wilson’s disease, and idiopathic (up to 14% of causes). Due to the many complications of cirrhosis, patient life expectancy is drastically reduced.
Major complications of cirrhosis include variceal hemorrhage, ascites, spontaneous bacterial peritonitis (SBP), hepatic encephalopathy, hepatorenal syndrome, and hepatopulmonary syndrome. If one of these develops, the patient is considered to have decompensated cirrhosis. Bleeding, infection, increased alcohol intake, dehydration, electrolyte abnormalities (hypokalemia specifically), and constipation can all ultimately contribute to the development of decompensated cirrhosis.
Pathologic changes in the liver lead to the above complications. These pathologic changes include fibrosis and the formation of regenerative nodules that replace normal hepatic tissue. This decreases hepatic venous flow and elevates portal pressures. This in turn causes splenomegaly, resulting in anemia, hypoalbuminemia, thrombocytopenia, and often ascites."


http://www.emdocs.net/approach-to-the-sick-cirrhotic-patient/